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Oxycodone Wiki2Web Clarity Challenge

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Study Guide: Oxycodone: Pharmacology, History, and Clinical Use

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Oxycodone: Pharmacology, History, and Clinical Use Study Guide

Historical Development and Early Use

Oxycodone was first synthesized in Germany in 1916, derived from thebaine.

Answer: True

Explanation: The synthesis of oxycodone from thebaine occurred in Germany in 1916.

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The first documented medical use of oxycodone occurred in Germany in 1917.

Answer: True

Explanation: Following its synthesis in 1916, oxycodone saw its first documented medical application in Germany in 1917.

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Skophedal, containing oxycodone, was used by the Wehrmacht and was noted for causing less sedation than other opiates at similar doses.

Answer: True

Explanation: Skophedal, a formulation containing oxycodone, was utilized by the Wehrmacht and was recognized for its potent analgesic effects with reportedly less sedation compared to other opiates at equivalent doses.

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Oxycodone was first synthesized in which country?

Answer: Germany

Explanation: The initial synthesis of oxycodone took place in Germany.

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The drug Skophedal, mentioned in the source, contained oxycodone and was used by which group?

Answer: The Wehrmacht (German Army) as a battlefield analgesic

Explanation: Skophedal, a formulation containing oxycodone, was employed by the Wehrmacht as a battlefield analgesic.

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What does the 'Miracle Drug of the 1930s' designation for oxycodone in Europe signify?

Answer: It was perceived as highly effective and well-received during that decade.

Explanation: The designation 'Miracle Drug of the 1930s' in Europe reflects the perception of oxycodone as highly effective and widely accepted during that period.

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Pharmacological Profile

Oxycodone functions by blocking the mu-opioid receptor (MOR) to prevent pain signals.

Answer: False

Explanation: Oxycodone acts as an agonist, activating the mu-opioid receptor (MOR), rather than blocking it.

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Oxycodone and codeine share the exact same chemical structure.

Answer: False

Explanation: Oxycodone and codeine possess distinct chemical structures; oxycodone has specific modifications, such as a hydroxyl group at carbon-14, differentiating it from codeine.

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The IUPAC name for oxycodone is a relatively simple chemical descriptor.

Answer: False

Explanation: The IUPAC name for oxycodone, (5R,9R,13S,14S)-4,5α-Epoxy-14-hydroxy-3-methoxy-17-methylmorphinan-6-one, is a complex and precise chemical descriptor.

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What is the primary mechanism of action for oxycodone?

Answer: Activating the mu-opioid receptor (MOR).

Explanation: Oxycodone's primary mechanism of action involves the activation of the mu-opioid receptor (MOR).

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Which structural modification distinguishes oxycodone from codeine, as per the source?

Answer: Oxycodone has a hydroxyl group at carbon-14.

Explanation: A key structural distinction is the presence of a hydroxyl group at carbon-14 in oxycodone, which is absent in codeine.

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Pharmacokinetics and Metabolism

The primary enzyme responsible for the O-demethylation of oxycodone is CYP3A4.

Answer: False

Explanation: The O-demethylation of oxycodone to oxymorphone is primarily mediated by the CYP2D6 enzyme, not CYP3A4.

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Noroxycodone and oxymorphone are identified as the main metabolites of oxycodone, both possessing pharmacological activity.

Answer: True

Explanation: Noroxycodone and oxymorphone are indeed the principal metabolites of oxycodone, and both exhibit pharmacological activity.

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When taken orally, oxycodone is less potent than morphine.

Answer: False

Explanation: Orally administered oxycodone is approximately 1.5 times more potent than oral morphine, not less potent.

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The bioavailability of oral oxycodone typically ranges between 60% and 87%.

Answer: True

Explanation: The oral bioavailability of oxycodone generally falls within the range of 60% to 87%.

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Oxycodone and its metabolites cannot be detected in biological fluids like blood or urine.

Answer: False

Explanation: Oxycodone and its metabolites are detectable in biological fluids such as blood and urine, which is utilized in drug monitoring and forensic analysis.

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Oxymorphone, an active metabolite of oxycodone, has a lower affinity for the mu-opioid receptor compared to oxycodone.

Answer: False

Explanation: Oxymorphone, an active metabolite of oxycodone, possesses a higher affinity for the mu-opioid receptor than oxycodone itself.

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The elimination half-life of immediate-release oxycodone is typically around 4.5 hours.

Answer: False

Explanation: The elimination half-life of immediate-release oxycodone is generally shorter, around 2-3 hours, whereas the controlled-release formulation has a half-life closer to 4.5 hours.

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Intranasal oxycodone administration shows significantly lower bioavailability than oral administration.

Answer: False

Explanation: Intranasal administration of oxycodone exhibits a bioavailability of approximately 77%, which is comparable to, not significantly lower than, oral administration.

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How does the oral analgesic potency of oxycodone compare to oral morphine?

Answer: Oxycodone is approximately 1.5 times more potent than morphine.

Explanation: When administered orally, oxycodone exhibits approximately 1.5 times the analgesic potency of morphine.

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Which liver enzymes are primarily responsible for the metabolism of oxycodone?

Answer: CYP3A4 and CYP2D6

Explanation: Oxycodone metabolism is primarily carried out by the hepatic enzymes CYP3A4 and CYP2D6.

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According to the source, what are the two main active metabolites of oxycodone?

Answer: Noroxycodone and Oxymorphone

Explanation: The principal active metabolites of oxycodone are noroxycodone and oxymorphone.

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What is the typical bioavailability range for orally administered oxycodone?

Answer: 60% - 87%

Explanation: The bioavailability of oxycodone following oral administration typically ranges between 60% and 87%.

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How does the potency of intravenous oxycodone compare to intravenous morphine?

Answer: They are considered equianalgesic (1:1 ratio).

Explanation: Intravenously administered oxycodone and morphine are considered equianalgesic, meaning they possess similar potency at a 1:1 ratio.

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What is the approximate bioavailability of intranasally administered oxycodone?

Answer: Approximately 77%

Explanation: Intranasal administration of oxycodone yields an approximate bioavailability of 77%.

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Clinical Use, Formulations, and Administration

Oxycodone is primarily indicated for the treatment of mild pain and inflammation.

Answer: False

Explanation: The primary indication for oxycodone is for moderate to severe pain, not mild pain or inflammation.

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OxyContin is a brand name for an immediate-release formulation of oxycodone.

Answer: False

Explanation: OxyContin is specifically an extended-release formulation of oxycodone, designed for prolonged pain management.

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Oxycodone is sometimes combined with naloxone to improve its pain-relieving properties.

Answer: False

Explanation: When combined with oxycodone, naloxone is typically included to deter abuse and counteract constipation, not to enhance pain relief.

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Immediate-release oxycodone formulations are designed to provide pain relief for 10-12 hours.

Answer: False

Explanation: Immediate-release oxycodone formulations are designed for shorter durations, typically providing relief for 3-6 hours, not 10-12 hours.

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According to the source, oxycodone use in early pregnancy is considered definitively unsafe.

Answer: False

Explanation: The source indicates that oxycodone use in early pregnancy appears relatively safe, though consultation with a healthcare provider is always advised.

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What is the primary medical purpose of oxycodone as stated in the source?

Answer: To manage moderate to severe acute or chronic pain when other treatments are insufficient.

Explanation: The source material explicitly states that oxycodone is primarily indicated for the management of moderate to severe acute or chronic pain when alternative treatment modalities are inadequate.

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Which of the following is identified as an extended-release formulation of oxycodone?

Answer: OxyContin

Explanation: OxyContin is identified as an extended-release formulation of oxycodone, designed for prolonged pain management.

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What is the typical duration of pain relief provided by immediate-release oxycodone?

Answer: 3 to 6 hours

Explanation: Immediate-release oxycodone formulations typically provide pain relief for a duration of 3 to 6 hours.

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When oxycodone is combined with naloxone in certain formulations, what is the purpose of the naloxone?

Answer: To deter abuse and counteract constipation.

Explanation: In combination formulations, naloxone serves to deter abuse and mitigate opioid-induced constipation.

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The 'Contradicts other' notice in the pharmacology section indicates a potential issue with:

Answer: Discrepancies in equianalgesic data between articles.

Explanation: The 'Contradicts other' notation highlights potential discrepancies in equianalgesic data presented across different sections or related articles.

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In which region is oxycodone mentioned as being administered via injection?

Answer: United Kingdom

Explanation: The source mentions that oxycodone can be administered via injection in the United Kingdom.

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Adverse Effects, Dependence, and Abuse

Oxycodone possesses a low potential for addiction and abuse, rendering it a relatively safe medication.

Answer: False

Explanation: Oxycodone has a high potential for addiction and abuse, classifying it as a controlled substance with significant risks.

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Common side effects associated with oxycodone use include euphoria, nausea, and drowsiness.

Answer: True

Explanation: Euphoria, nausea, and drowsiness are indeed among the frequently observed side effects of oxycodone administration.

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Abruptly discontinuing oxycodone after developing physical dependence poses a high risk of severe withdrawal symptoms.

Answer: True

Explanation: Sudden cessation of oxycodone in physically dependent individuals can precipitate severe and distressing withdrawal symptoms.

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Chronic oxycodone use can lead to hypogonadism, characterized by reduced sex hormone levels.

Answer: True

Explanation: Chronic administration of oxycodone is associated with hypogonadism, a condition marked by diminished levels of sex hormones.

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Symptoms of an oxycodone overdose include rapid breathing and increased heart rate.

Answer: False

Explanation: Symptoms of an oxycodone overdose typically involve slowed or shallow breathing and a decreased heart rate, not rapid breathing and increased heart rate.

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The primary danger of chronic constipation from oxycodone is mild discomfort.

Answer: False

Explanation: The primary danger associated with chronic constipation induced by oxycodone is not mild discomfort but the potential for severe complications, including bowel perforations.

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Which of the following is listed as a common side effect of oxycodone?

Answer: Itching

Explanation: Itching (pruritus) is frequently listed as a common side effect associated with oxycodone use.

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What is a significant risk if a patient physically dependent on oxycodone stops taking it abruptly?

Answer: Severe withdrawal symptoms.

Explanation: Abrupt cessation of oxycodone in physically dependent individuals carries a significant risk of precipitating severe withdrawal symptoms.

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Chronic use of oxycodone can potentially lead to which hormonal imbalance?

Answer: Hypogonadism

Explanation: Chronic oxycodone use is associated with the potential development of hypogonadism, characterized by reduced sex hormone levels.

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Which of the following is NOT a symptom of oxycodone overdose according to the source?

Answer: Rapid, deep breathing

Explanation: Rapid, deep breathing is not characteristic of an oxycodone overdose; symptoms typically include shallow breathing, slowed heart rate, and constricted pupils.

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Which of the following is categorized as a less common but potentially serious side effect of oxycodone?

Answer: Delirium

Explanation: Delirium is considered a less common but potentially serious adverse effect associated with oxycodone use.

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What is the primary danger associated with chronic constipation induced by oxycodone?

Answer: It can cause severe complications like bowel perforations.

Explanation: Chronic constipation resulting from oxycodone use poses a significant danger, with the potential for severe complications such as bowel perforations.

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How does oxycodone's activation of the mu-opioid receptor (MOR) contribute to addiction?

Answer: By activating MOR in the mesolimbic reward pathway.

Explanation: Activation of the mu-opioid receptor (MOR) within the mesolimbic reward pathway by oxycodone is implicated in its contribution to addiction.

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The development of tolerance to oxycodone's effects is described as a complex process involving:

Answer: Receptor-level, cellular-level, and system-level neural adaptations.

Explanation: Tolerance to oxycodone develops through a complex interplay of adaptations at receptor, cellular, and system-level neural networks.

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Regulatory Landscape and Societal Impact

In the United States, oxycodone is classified as a Schedule II controlled substance.

Answer: True

Explanation: Oxycodone is indeed classified as a Schedule II controlled substance within the United States regulatory framework.

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Purdue Pharma's development of OxyContin significantly contributed to the opioid epidemic.

Answer: True

Explanation: The marketing and widespread use of OxyContin by Purdue Pharma are widely recognized as major contributors to the opioid epidemic in the United States.

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The 1961 Single Convention on Narcotic Drugs classified oxycodone under Schedule I, imposing strict international controls.

Answer: True

Explanation: The 1961 Single Convention on Narcotic Drugs classified oxycodone under Schedule I, establishing stringent international controls on its use.

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Purdue Pharma's reformulation of OxyContin aimed to make the pills more resistant to crushing and dissolving to reduce abuse.

Answer: True

Explanation: Purdue Pharma reformulated OxyContin with the objective of increasing resistance to crushing and dissolution, thereby aiming to mitigate abuse potential.

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The FDA's 2013 labeling update for long-acting opioids removed the indication for moderate pain.

Answer: True

Explanation: The FDA's 2013 labeling update for long-acting opioids did indeed remove the indication for moderate pain, restricting its use to severe pain.

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What is the legal classification of oxycodone in the United States?

Answer: Schedule II

Explanation: In the United States, oxycodone is classified as a Schedule II controlled substance.

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Purdue Pharma is primarily associated with the development and marketing of which oxycodone formulation?

Answer: OxyContin

Explanation: Purdue Pharma is principally recognized for the development and marketing of OxyContin, an extended-release formulation of oxycodone.

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The 1961 Single Convention on Narcotic Drugs classified oxycodone under which schedule?

Answer: Schedule I

Explanation: The 1961 Single Convention on Narcotic Drugs placed oxycodone under Schedule I, signifying strict international controls.

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According to the source, since 2012, which drugs have become more prominent causes of drug-related deaths in the US compared to oxycodone?

Answer: Heroin and Fentanyl

Explanation: Since 2012, heroin and fentanyl have emerged as more prominent causes of drug-related deaths in the US relative to oxycodone.

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What was the main goal of the FDA's 2013 labeling update for long-acting opioids?

Answer: To restrict use to severe, long-term pain management.

Explanation: The primary objective of the FDA's 2013 labeling update for long-acting opioids was to restrict their indication to severe, long-term pain management.

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What does the source indicate about the non-medical use of oxycodone among injecting drug users in Australia by 2015?

Answer: A significant majority (91%) reported using it, with notable recent injection.

Explanation: By 2015, the source indicates that a substantial majority (91%) of injecting drug users in Australia reported oxycodone use, with a notable prevalence of recent injection.

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