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Zafirlukast: Pharmacology and Clinical Application

At a Glance

Title: Zafirlukast: Pharmacology and Clinical Application

Total Categories: 6

Category Stats

  • Pharmacological Profile and Mechanism of Action: 6 flashcards, 8 questions
  • Clinical Indications, Contraindications, and Therapeutic Use: 5 flashcards, 9 questions
  • Pharmacokinetics, Metabolism, and Elimination: 14 flashcards, 18 questions
  • Adverse Effects, Safety, and Drug Interactions: 8 flashcards, 13 questions
  • Special Populations, Administration, and Storage: 7 flashcards, 11 questions
  • Chemical Properties, Identifiers, and Historical Context: 10 flashcards, 16 questions

Total Stats

  • Total Flashcards: 50
  • True/False Questions: 48
  • Multiple Choice Questions: 27
  • Total Questions: 75

Instructions

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Welcome to Your Curriculum Command Center

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The Core Concept: What is a "Kit"?

Think of a Kit as your all-in-one digital lesson plan. It's a single, portable file that contains every piece of content for a topic: your subject categories, a central image, all your flashcards, and all your questions. The true power of the Studio is speed—once a kit is made (or you import one), you are just minutes away from printing an entire set of coursework.

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Step 1: Laying the Foundation (The Authoring Tools)

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⚙️ Kit Manager: Your Kit's Identity

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Study Guide: Zafirlukast: Pharmacology and Clinical Application

Study Guide: Zafirlukast: Pharmacology and Clinical Application

Pharmacological Profile and Mechanism of Action

Zafirlukast functions by directly relaxing the smooth muscles in the airways to improve airflow.

Answer: False

Zafirlukast functions by blocking the action of cysteinyl leukotrienes on CysLT1 receptors, which reduces airway inflammation and bronchoconstriction, rather than by directly relaxing smooth muscles.

Related Concepts:

  • What is the pharmacodynamic action of Zafirlukast?: Pharmacodynamically, Zafirlukast acts as an antagonist of the cysteinyl leukotriene receptor 1 (CysLT1). This receptor is found on smooth muscle in the lungs, lung macrophages, and epithelial cells, and its activation by leukotrienes contributes to asthma symptoms like bronchoconstriction.

Zafirlukast's pharmacodynamic action involves blocking the effects of cysteinyl leukotrienes on specific receptors.

Answer: True

Pharmacodynamically, Zafirlukast acts as an antagonist of the cysteinyl leukotriene receptor 1 (CysLT1), blocking the effects of cysteinyl leukotrienes.

Related Concepts:

  • What is the pharmacodynamic action of Zafirlukast?: Pharmacodynamically, Zafirlukast acts as an antagonist of the cysteinyl leukotriene receptor 1 (CysLT1). This receptor is found on smooth muscle in the lungs, lung macrophages, and epithelial cells, and its activation by leukotrienes contributes to asthma symptoms like bronchoconstriction.

Cysteinyl leukotrienes contribute to asthma symptoms by causing vasodilation and reducing mucus secretion.

Answer: False

Cysteinyl leukotrienes contribute to asthma symptoms by causing bronchoconstriction, increasing mucus secretion, and promoting airway edema, not vasodilation.

Related Concepts:

  • What is the role of cysteinyl leukotrienes in asthma, and how does Zafirlukast counteract them?: Cysteinyl leukotrienes (LTC4, LTD4, LTE4), produced by inflammatory cells like eosinophils, trigger bronchoconstriction, mucus secretion, and edema in the airways, contributing to asthma symptoms. Zafirlukast blocks the action of these leukotrienes by binding to their CysLT1 receptor, thereby exerting an anti-inflammatory effect.

Zafirlukast antagonizes cysteinyl leukotrienes, including LTC4, LTD4, and LTE4.

Answer: True

Zafirlukast acts as an antagonist to cysteinyl leukotrienes, specifically Leukotriene C4 (LTC4), Leukotriene D4 (LTD4), and Leukotriene E4 (LTE4).

Related Concepts:

  • What specific leukotrienes does Zafirlukast antagonize?: Zafirlukast antagonizes cysteinyl leukotrienes, specifically Leukotriene C4 (LTC4), Leukotriene D4 (LTD4), and Leukotriene E4 (LTE4).

Research suggests Zafirlukast may act as a partial inverse agonist at the CysLT1 receptor, though its primary role is antagonist.

Answer: True

Some research indicates Zafirlukast might function as a partial inverse agonist at the CysLT1 receptor, in addition to its primary role as an antagonist.

Related Concepts:

  • What is the potential mechanism of action of Zafirlukast beyond being a simple antagonist?: There is some research suggesting that Zafirlukast might act as a partial inverse agonist at the CysLT1 receptor, although its primary classification remains as an antagonist. The clinical significance of this potential inverse agonism is currently unknown.

What is the primary classification of Zafirlukast?

Answer: A leukotriene receptor antagonist (LTRA)

Zafirlukast is primarily classified as an orally administered leukotriene receptor antagonist (LTRA).

Related Concepts:

  • What is Zafirlukast and what is its primary medical use?: Zafirlukast is an orally administered medication classified as a leukotriene receptor antagonist (LTRA). Its primary medical use is for the chronic treatment of asthma in adults and children over five years old.

How does Zafirlukast exert its therapeutic effect in asthma?

Answer: By blocking the action of cysteinyl leukotrienes on CysLT1 receptors

Zafirlukast exerts its therapeutic effect by competitively inhibiting the binding of cysteinyl leukotrienes (LTC4, LTD4, LTE4) to their CysLT1 receptors on airway cells.

Related Concepts:

What is the primary role of cysteinyl leukotrienes in asthma, according to the source?

Answer: To cause bronchoconstriction, mucus secretion, and edema

Cysteinyl leukotrienes play a primary role in asthma by inducing bronchoconstriction, increasing mucus secretion, and contributing to airway edema.

Related Concepts:

  • What is the role of cysteinyl leukotrienes in asthma, and how does Zafirlukast counteract them?: Cysteinyl leukotrienes (LTC4, LTD4, LTE4), produced by inflammatory cells like eosinophils, trigger bronchoconstriction, mucus secretion, and edema in the airways, contributing to asthma symptoms. Zafirlukast blocks the action of these leukotrienes by binding to their CysLT1 receptor, thereby exerting an anti-inflammatory effect.

Clinical Indications, Contraindications, and Therapeutic Use

Zafirlukast is primarily prescribed for the immediate relief of acute asthma attacks.

Answer: False

Zafirlukast is indicated for the chronic management of asthma and is not designed for the immediate relief of acute asthma attacks.

Related Concepts:

  • Can Zafirlukast be used to treat an acute asthma attack?: No, Zafirlukast is not effective in the event of an acute asthma attack. It is intended for the long-term, chronic management of asthma symptoms.

Zafirlukast is considered more effective than inhaled glucocorticoids when used as the sole treatment for asthma.

Answer: False

Zafirlukast is generally considered less effective than inhaled glucocorticoids when used as monotherapy for asthma management.

Related Concepts:

  • How does Zafirlukast compare in effectiveness to other asthma treatments like inhaled glucocorticoids?: Zafirlukast is generally considered less effective than inhaled glucocorticoids when used as monotherapy for asthma. It is also typically less effective than long-acting beta-2 agonists when used in combination therapy, though it is useful for long-term asthma treatment.

Zafirlukast is recommended for patients with a known hypersensitivity to the drug.

Answer: False

Zafirlukast is contraindicated in individuals who have a known hypersensitivity or allergic reaction to the medication.

Related Concepts:

  • Under what circumstances is Zafirlukast contraindicated?: Zafirlukast is contraindicated in individuals who have a known hypersensitivity or allergic reaction to the medication.

Zafirlukast has demonstrated clear cost-effectiveness in managing asthma.

Answer: False

While some preliminary evidence suggests potential cost savings, the overall cost-effectiveness of Zafirlukast in asthma management has not been definitively established.

Related Concepts:

  • What is the current understanding of Zafirlukast's cost-effectiveness?: While preliminary evidence suggests Zafirlukast might reduce healthcare costs, its overall cost-effectiveness has not been definitively established.

Zafirlukast has been investigated for potential benefits in treating chronic urticaria and COPD.

Answer: True

Research has explored Zafirlukast's potential therapeutic benefits beyond asthma, including its investigation for treating chronic urticaria and improving lung function in patients with Chronic Obstructive Pulmonary Disease (COPD).

Related Concepts:

  • What other indications, besides asthma, have been investigated or suggested for Zafirlukast?: Research suggests Zafirlukast may be beneficial for chronic urticaria (hives), cystic fibrosis (based on a pilot study), and has shown improvement in lung function in patients with Chronic Obstructive Pulmonary Disease (COPD).

For which condition is Zafirlukast primarily indicated?

Answer: Chronic management of asthma in adults and children over five years old

Zafirlukast is primarily indicated for the chronic management of asthma in adults and children aged five years and older.

Related Concepts:

  • What is Zafirlukast and what is its primary medical use?: Zafirlukast is an orally administered medication classified as a leukotriene receptor antagonist (LTRA). Its primary medical use is for the chronic treatment of asthma in adults and children over five years old.

Zafirlukast is generally considered less effective than which other class of asthma medications when used alone?

Answer: Inhaled glucocorticoids

When used as monotherapy for asthma, Zafirlukast is generally considered less effective than inhaled glucocorticoids.

Related Concepts:

  • How does Zafirlukast compare in effectiveness to other asthma treatments like inhaled glucocorticoids?: Zafirlukast is generally considered less effective than inhaled glucocorticoids when used as monotherapy for asthma. It is also typically less effective than long-acting beta-2 agonists when used in combination therapy, though it is useful for long-term asthma treatment.

Zafirlukast is contraindicated in patients with:

Answer: Known hypersensitivity to the medication

Zafirlukast is contraindicated in patients who exhibit known hypersensitivity or allergic reactions to the drug itself.

Related Concepts:

  • Under what circumstances is Zafirlukast contraindicated?: Zafirlukast is contraindicated in individuals who have a known hypersensitivity or allergic reaction to the medication.

Which of the following is NOT mentioned as an investigated or suggested indication for Zafirlukast besides asthma?

Answer: Hypertension

While Zafirlukast has been investigated for chronic urticaria and COPD, hypertension is not mentioned as an investigated or suggested indication.

Related Concepts:

  • What other indications, besides asthma, have been investigated or suggested for Zafirlukast?: Research suggests Zafirlukast may be beneficial for chronic urticaria (hives), cystic fibrosis (based on a pilot study), and has shown improvement in lung function in patients with Chronic Obstructive Pulmonary Disease (COPD).

Pharmacokinetics, Metabolism, and Elimination

Kidney impairment significantly affects the way Zafirlukast is absorbed and eliminated by the body.

Answer: False

Renal impairment does not appear to significantly alter the pharmacokinetic profile of Zafirlukast, meaning its absorption, distribution, metabolism, and excretion are not notably affected by kidney issues.

Related Concepts:

  • Does renal impairment affect how Zafirlukast is processed by the body?: No, renal impairment does not appear to significantly alter the pharmacokinetic profile of Zafirlukast, meaning its absorption, distribution, metabolism, and excretion are not notably affected by kidney issues.

Significant liver damage can lead to increased clearance of Zafirlukast from the body.

Answer: False

Significant hepatic impairment can impair the clearance of Zafirlukast, potentially leading to increased plasma concentrations and drug exposure, rather than increased clearance.

Related Concepts:

  • How does hepatic impairment influence Zafirlukast pharmacokinetics?: Significant hepatic impairment can impair the clearance of Zafirlukast. In patients with cirrhosis, for example, the maximum plasma concentration and overall drug exposure can increase by 50-60%.

Zafirlukast is extensively metabolized in the liver by the enzyme CYP2C9 into active metabolites.

Answer: False

Zafirlukast is extensively metabolized in the liver by CYP2C9, but its metabolites are considered inactive.

Related Concepts:

  • How is Zafirlukast metabolized in the body?: Zafirlukast undergoes extensive metabolism in the liver, primarily mediated by the enzyme CYP2C9. This process converts Zafirlukast into inactive hydroxylated metabolites.

The primary route of elimination for Zafirlukast from the body is through urine.

Answer: False

The primary route of elimination for Zafirlukast is through biliary excretion, leading to fecal elimination; it is not significantly excreted via urine.

Related Concepts:

  • What is the primary route of elimination for Zafirlukast from the body?: Zafirlukast is primarily eliminated through biliary excretion. It is not detectable in urine, and its mean terminal half-life ranges from 8 to 16 hours.

Genetic variations in the LTC4 synthase gene are linked to a potentially better response to Zafirlukast.

Answer: False

Genetic polymorphisms in the LTC4 synthase gene have been associated with a decreased clinical response to Zafirlukast, particularly in individuals with severe asthma phenotypes.

Related Concepts:

  • How might genetic variations in LTC4 synthase affect a person's response to Zafirlukast?: Genetic polymorphisms in the LTC4 synthase promoter, such as the A444C single-nucleotide polymorphism (SNP), have been associated with a decreased clinical response to Zafirlukast, particularly in individuals with severe asthma phenotypes.

Individuals with genetic variations that reduce CYP2C9 function may experience lower Zafirlukast levels.

Answer: False

Genetic variations that reduce CYP2C9 function can lead to decreased clearance of Zafirlukast, consequently resulting in higher plasma levels and increased drug exposure.

Related Concepts:

  • What is the significance of CYP2C9 genetic variations in relation to Zafirlukast treatment?: Since Zafirlukast is metabolized by CYP2C9, genetic variations (SNPs) that reduce CYP2C9 function, like CYP2C9*3 or CYP2C9*13, can lead to decreased clearance of the drug and consequently increased exposure. The CYP2C9*3 variant is more prevalent in individuals of South/Central Asian ancestry.

Zafirlukast's interaction with CYP2C9 primarily involves its inhibition of this enzyme, thereby affecting the metabolism of other drugs.

Answer: False

Zafirlukast is primarily metabolized by CYP2C9, meaning it is a substrate for this enzyme. Its primary interaction affecting other drugs involves the inhibition of CYP3A4, not CYP2C9.

Related Concepts:

  • How does Zafirlukast's interaction with CYP2C9 affect drug metabolism?: Zafirlukast itself is metabolized by CYP2C9. This means that other drugs that are also metabolized by CYP2C9 might have their metabolism affected by Zafirlukast, potentially leading to altered drug levels in the body.

The bioavailability of Zafirlukast is well-documented and known to be high.

Answer: False

The bioavailability of Zafirlukast is listed as unknown in the provided source material.

Related Concepts:

  • What is the bioavailability of Zafirlukast?: The bioavailability of Zafirlukast is listed as unknown in the provided source.

Zafirlukast is highly bound to plasma proteins, primarily albumin, exceeding 99% binding.

Answer: True

Zafirlukast exhibits high protein binding in plasma, with over 99% bound to albumin.

Related Concepts:

  • What is the protein binding percentage for Zafirlukast, and to which protein is it primarily bound?: Zafirlukast is highly protein-bound, with over 99% of it bound to albumin, the most abundant protein in human plasma.

The primary metabolites of Zafirlukast are pharmacologically active and contribute to its therapeutic effect.

Answer: False

The primary metabolites of Zafirlukast are hydroxylated and considered pharmacologically inactive.

Related Concepts:

  • What are the known metabolites of Zafirlukast?: Zafirlukast is metabolized into hydroxylated metabolites, which are considered inactive.

Zafirlukast has an elimination half-life of approximately 24 hours.

Answer: False

The elimination half-life of Zafirlukast is approximately 10 hours, not 24 hours.

Related Concepts:

  • What is the elimination half-life of Zafirlukast?: The elimination half-life of Zafirlukast is approximately 10 hours.

Fecal excretion is the main pathway for Zafirlukast elimination from the body.

Answer: True

Zafirlukast is primarily eliminated through biliary excretion, which results in fecal excretion being the main pathway from the body.

Related Concepts:

  • What is the primary route of excretion for Zafirlukast?: Zafirlukast is primarily excreted through feces.

Albumin's role concerning Zafirlukast is primarily related to its metabolism in the liver.

Answer: False

Albumin's primary role concerning Zafirlukast is its high protein binding, facilitating transport throughout the body, not its metabolism in the liver.

Related Concepts:

  • What is the role of albumin in drug distribution, as relevant to Zafirlukast?: Albumin is the most abundant protein in human plasma and plays a crucial role in transporting substances, including drugs like Zafirlukast, throughout the body due to its high binding capacity.

How does hepatic impairment affect Zafirlukast pharmacokinetics?

Answer: It can impair clearance, leading to increased plasma concentration.

Significant hepatic impairment can impair the clearance of Zafirlukast, leading to an increase in its plasma concentration and overall drug exposure.

Related Concepts:

  • How does hepatic impairment influence Zafirlukast pharmacokinetics?: Significant hepatic impairment can impair the clearance of Zafirlukast. In patients with cirrhosis, for example, the maximum plasma concentration and overall drug exposure can increase by 50-60%.

Which enzyme is primarily responsible for the metabolism of Zafirlukast?

Answer: CYP2C9

Zafirlukast undergoes extensive metabolism in the liver, primarily mediated by the cytochrome P450 enzyme CYP2C9.

Related Concepts:

  • How is Zafirlukast metabolized in the body?: Zafirlukast undergoes extensive metabolism in the liver, primarily mediated by the enzyme CYP2C9. This process converts Zafirlukast into inactive hydroxylated metabolites.

What is the primary route of elimination for Zafirlukast?

Answer: Biliary excretion

The primary route of elimination for Zafirlukast from the body is through biliary excretion, leading to its presence in feces.

Related Concepts:

  • What is the primary route of excretion for Zafirlukast?: Zafirlukast is primarily excreted through feces.

Zafirlukast is highly bound to which plasma protein?

Answer: Albumin

Zafirlukast is highly bound to plasma proteins, predominantly albumin, with binding exceeding 99%.

Related Concepts:

  • What is the protein binding percentage for Zafirlukast, and to which protein is it primarily bound?: Zafirlukast is highly protein-bound, with over 99% of it bound to albumin, the most abundant protein in human plasma.

The source states that Zafirlukast's bioavailability is:

Answer: Unknown

According to the provided source material, the bioavailability of Zafirlukast is listed as unknown.

Related Concepts:

  • What is the bioavailability of Zafirlukast?: The bioavailability of Zafirlukast is listed as unknown in the provided source.

Adverse Effects, Safety, and Drug Interactions

Headaches and nausea are the most frequently reported side effects of Zafirlukast.

Answer: True

Headaches and gastrointestinal upset, including nausea, are among the most frequently reported side effects associated with Zafirlukast use.

Related Concepts:

  • What are the common side effects associated with Zafirlukast use?: The most common side effects of Zafirlukast include headaches and gastrointestinal upset, such as nausea, stomach discomfort, and diarrhea. Headaches occur with a frequency similar to placebo in clinical trials.

Hallucinations and insomnia are considered rare side effects of Zafirlukast.

Answer: False

While hallucinations and insomnia are potential neuropsychiatric side effects associated with Zafirlukast and other leukotriene receptor antagonists, they are not exclusively classified as rare. They are part of a spectrum of potential neuropsychiatric effects identified through post-marketing surveillance.

Related Concepts:

  • What are the potential neuropsychiatric side effects associated with Zafirlukast and other LTRAs?: Neuropsychiatric side effects, ranging from less severe issues like abnormal dreams to more serious ones like hallucinations, tremors, and suicidality, have been reported with Zafirlukast and other LTRAs. These were identified through post-marketing surveillance as initial trials were not specifically designed to detect them.

Neuropsychiatric side effects associated with Zafirlukast were primarily identified during initial clinical trials.

Answer: False

Neuropsychiatric side effects associated with Zafirlukast were primarily identified through post-marketing surveillance, as initial clinical trials were not specifically designed to detect these effects.

Related Concepts:

  • What are the potential neuropsychiatric side effects associated with Zafirlukast and other LTRAs?: Neuropsychiatric side effects, ranging from less severe issues like abnormal dreams to more serious ones like hallucinations, tremors, and suicidality, have been reported with Zafirlukast and other LTRAs. These were identified through post-marketing surveillance as initial trials were not specifically designed to detect them.

Hepatotoxicity, or liver damage, from Zafirlukast typically occurs within the first month of treatment.

Answer: False

Zafirlukast-induced hepatotoxicity typically occurs within 2 to 6 months of initiating treatment, not usually within the first month.

Related Concepts:

  • What is hepatotoxicity, and how can it manifest with Zafirlukast use?: Hepatotoxicity refers to liver damage. With Zafirlukast, it can manifest as rare but serious acute liver injury, typically occurring within 2-6 months of starting the medication. Symptoms include fatigue, nausea, dark urine, jaundice, and itching, often with elevated liver enzymes resembling viral hepatitis.

The exact mechanism causing Zafirlukast-induced hepatotoxicity is well understood and confirmed.

Answer: False

The precise mechanism underlying Zafirlukast-induced hepatotoxicity remains unclear and is hypothesized to involve a metabolic intermediate.

Related Concepts:

  • What is the suspected mechanism behind Zafirlukast-induced hepatotoxicity?: The exact mechanism is unclear, but it is hypothesized that hepatotoxicity may be caused by a metabolic intermediate of Zafirlukast, as the drug is metabolized in the liver by the enzyme CYP2C9.

Eosinophilic Granulomatosis with Polyangiitis (EGPA) is directly caused by Zafirlukast treatment.

Answer: False

While cases of EGPA have been reported in patients using Zafirlukast, it is not believed to be directly causative but rather associated with underlying conditions and treatment changes in asthma management.

Related Concepts:

  • What is the relationship between Zafirlukast and Eosinophilic Granulomatosis with Polyangiitis (EGPA)?: Several cases of EGPA have been reported in patients using Zafirlukast and other asthma medications. While an association exists, it is not believed to be directly causative; EGPA often appears in patients with long-standing asthma and sinus inflammation, particularly when chronic corticosteroids are tapered after initiating new asthma therapy like Zafirlukast.

Overdoses of Zafirlukast, even up to 200 mg, have consistently resulted in fatalities.

Answer: False

The highest reported overdose amount for Zafirlukast was 200 mg, and all patients who experienced overdoses survived, reporting symptoms such as rash and upset stomach.

Related Concepts:

  • What is the highest reported overdose amount for Zafirlukast, and what were the outcomes?: The highest reported overdose amount for Zafirlukast was 200 mg. All patients who experienced overdoses survived, reporting symptoms such as rash and upset stomach.

Zafirlukast can increase the concentration of drugs metabolized by CYP3A4, such as warfarin.

Answer: True

Zafirlukast inhibits the cytochrome P450 enzyme CYP3A4, which can lead to increased concentrations of other drugs metabolized by this enzyme, such as warfarin.

Related Concepts:

  • How does Zafirlukast interact with other drugs metabolized by CYP3A4?: Zafirlukast inhibits the cytochrome P450 enzyme CYP3A4. This inhibition can lead to increased concentrations of other drugs that are also metabolized by CYP3A4, potentially causing drug-drug interactions. Examples include the anticoagulant warfarin and antiepileptic drugs like phenytoin and carbamazepine.

Zafirlukast-induced hepatotoxicity predominantly affects males and is usually mild.

Answer: False

Zafirlukast-induced hepatotoxicity predominantly affects females and can range from mild to fatal, posing a significant risk.

Related Concepts:

  • What are the potential risks associated with Zafirlukast-induced hepatotoxicity, and who is predominantly affected?: Zafirlukast-induced hepatotoxicity can be fatal and may lead to more severe injury upon re-exposure. It has predominantly occurred in females. The liver damage pattern often resembles acute viral hepatitis.

Which of the following is a common side effect of Zafirlukast?

Answer: Headaches and gastrointestinal upset

Headaches and gastrointestinal upset, such as nausea and stomach discomfort, are among the most frequently reported side effects of Zafirlukast.

Related Concepts:

  • What are the common side effects associated with Zafirlukast use?: The most common side effects of Zafirlukast include headaches and gastrointestinal upset, such as nausea, stomach discomfort, and diarrhea. Headaches occur with a frequency similar to placebo in clinical trials.

Which of the following is a potential serious adverse effect associated with Zafirlukast?

Answer: Acute liver injury (hepatotoxicity)

Acute liver injury, or hepatotoxicity, is a potential serious adverse effect associated with Zafirlukast treatment.

Related Concepts:

  • What is hepatotoxicity, and how can it manifest with Zafirlukast use?: Hepatotoxicity refers to liver damage. With Zafirlukast, it can manifest as rare but serious acute liver injury, typically occurring within 2-6 months of starting the medication. Symptoms include fatigue, nausea, dark urine, jaundice, and itching, often with elevated liver enzymes resembling viral hepatitis.

What effect does Zafirlukast have on drugs metabolized by CYP3A4?

Answer: It decreases their metabolism, potentially increasing their levels.

Zafirlukast inhibits CYP3A4, which can decrease the metabolism of drugs processed by this enzyme, potentially leading to elevated plasma concentrations of those drugs.

Related Concepts:

  • How does Zafirlukast interact with other drugs metabolized by CYP3A4?: Zafirlukast inhibits the cytochrome P450 enzyme CYP3A4. This inhibition can lead to increased concentrations of other drugs that are also metabolized by CYP3A4, potentially causing drug-drug interactions. Examples include the anticoagulant warfarin and antiepileptic drugs like phenytoin and carbamazepine.

Which of the following is a potential neuropsychiatric side effect reported with Zafirlukast?

Answer: Auditory hallucinations

Auditory hallucinations are among the potential neuropsychiatric side effects that have been reported in association with Zafirlukast use.

Related Concepts:

  • What are the potential neuropsychiatric side effects associated with Zafirlukast and other LTRAs?: Neuropsychiatric side effects, ranging from less severe issues like abnormal dreams to more serious ones like hallucinations, tremors, and suicidality, have been reported with Zafirlukast and other LTRAs. These were identified through post-marketing surveillance as initial trials were not specifically designed to detect them.

Special Populations, Administration, and Storage

Adults and children aged 5 to 12 years old should both take the 20 mg dosage of Zafirlukast.

Answer: False

The recommended dosage for Zafirlukast differs by age group; adults and children over 12 years typically take 20 mg, while children aged 5 to 12 years are prescribed 10 mg.

Related Concepts:

  • What are the available dosage forms and strengths for Zafirlukast, and how do they differ based on age?: Zafirlukast is available in two dosage forms: a 20 mg tablet for adults and children over 12 years old, and a 10 mg tablet for children aged 5 to 12 years old.

Zafirlukast tablets should be stored in a cool, damp place to maintain their stability.

Answer: False

Zafirlukast tablets should be stored at room temperature, protected from direct sunlight and moisture, not in a damp environment.

Related Concepts:

  • What are the recommended storage conditions for Zafirlukast tablets?: Zafirlukast tablets should be stored at room temperature, protected from direct sunlight and moisture.

Older adults (65+) taking Zafirlukast may experience increased plasma concentrations of the drug due to impaired hepatic clearance.

Answer: True

In individuals aged 65 and older, Zafirlukast's hepatic clearance is impaired, leading to higher plasma concentrations and increased drug exposure.

Related Concepts:

  • How might Zafirlukast affect older adults (65 years and above)?: In adults aged 65 and older, Zafirlukast's hepatic clearance is impaired, leading to a 2 to 3-fold increase in maximum plasma concentration and total drug exposure (area under the curve). Older adults may also have an increased risk of infections, particularly lower respiratory tract infections, though these are not typically severe.

Zafirlukast is classified as a pregnancy category C drug, indicating potential risks to the fetus.

Answer: False

Zafirlukast is classified as pregnancy category B. Animal studies did not demonstrate teratogenicity at doses up to 20 times the maximum recommended human dose.

Related Concepts:

  • What is the pregnancy category assigned to Zafirlukast, and what do animal studies indicate?: Zafirlukast is classified as pregnancy category B. Animal studies showed no teratogenicity at doses up to 20 times the maximum recommended human dose, although spontaneous abortions occurred in cynomolgus monkeys at the highest tested dose, which was also maternally toxic.

There is extensive data available on the effects of Zafirlukast use during lactation.

Answer: False

Data regarding the effects of Zafirlukast use during lactation is limited, with only a small percentage of the maternal dose expected to transfer to the infant via breast milk.

Related Concepts:

  • What is known about Zafirlukast use during lactation?: Research on Zafirlukast use in breastfeeding women is limited. Manufacturer data suggests about 0.6% of the maternal dose may transfer to the infant via breast milk, but the effects on the infant are unknown.

Taking Zafirlukast with a high-fat meal enhances its oral absorption.

Answer: False

The oral absorption of Zafirlukast is reduced by approximately 40% when taken with meals high in fat or protein; it should be taken on an empty stomach.

Related Concepts:

  • How does food intake affect the absorption of Zafirlukast?: The oral absorption of Zafirlukast is reduced by approximately 40% when taken with meals high in fat or protein. To maximize absorption, it should be taken on an empty stomach, defined as one hour before or two hours after a meal.

The effectiveness of Zafirlukast in pediatric asthma is generally higher in children with more severe, atopic asthma.

Answer: False

Research suggests that Zafirlukast may be more effective in younger children with less atopic asthma, rather than those with more severe, atopic asthma.

Related Concepts:

  • What is the significance of the 'atopy' factor in pediatric asthma treatment with Zafirlukast?: Atopy refers to a predisposition to allergic conditions like asthma, hay fever, and eczema. Leukotriene receptor antagonists like Zafirlukast are generally found to be more effective in children who are younger and have less atopic asthma.

What dosage strength of Zafirlukast is recommended for children aged 5 to 12 years?

Answer: 10 mg

For pediatric patients aged 5 to 12 years, the recommended dosage strength of Zafirlukast is 10 mg.

Related Concepts:

  • What are the available dosage forms and strengths for Zafirlukast, and how do they differ based on age?: Zafirlukast is available in two dosage forms: a 20 mg tablet for adults and children over 12 years old, and a 10 mg tablet for children aged 5 to 12 years old.

What is the recommended way to take Zafirlukast concerning meals?

Answer: Take on an empty stomach, one hour before or two hours after meals.

To maximize absorption, Zafirlukast should be taken on an empty stomach, defined as one hour before or two hours after a meal, as food intake reduces absorption.

Related Concepts:

  • How does food intake affect the absorption of Zafirlukast?: The oral absorption of Zafirlukast is reduced by approximately 40% when taken with meals high in fat or protein. To maximize absorption, it should be taken on an empty stomach, defined as one hour before or two hours after a meal.

What is the reported pregnancy category for Zafirlukast?

Answer: Category B

Zafirlukast is classified under pregnancy category B, indicating that animal reproduction studies have not demonstrated a risk to the fetus, and there are no adequate studies in pregnant women.

Related Concepts:

  • What is the pregnancy category assigned to Zafirlukast, and what do animal studies indicate?: Zafirlukast is classified as pregnancy category B. Animal studies showed no teratogenicity at doses up to 20 times the maximum recommended human dose, although spontaneous abortions occurred in cynomolgus monkeys at the highest tested dose, which was also maternally toxic.

How does Zafirlukast's absorption change when taken with a meal high in fat?

Answer: Absorption decreases by approximately 40%.

Taking Zafirlukast with a meal high in fat or protein reduces its oral absorption by approximately 40%.

Related Concepts:

  • How does food intake affect the absorption of Zafirlukast?: The oral absorption of Zafirlukast is reduced by approximately 40% when taken with meals high in fat or protein. To maximize absorption, it should be taken on an empty stomach, defined as one hour before or two hours after a meal.

Chemical Properties, Identifiers, and Historical Context

Accolate is the brand name for Zafirlukast in the United States and was the first drug of its kind approved by the FDA.

Answer: True

Accolate is the United States brand name for Zafirlukast, and it holds historical significance as the first cysteinyl leukotriene receptor antagonist approved by the FDA.

Related Concepts:

  • What is the brand name for Zafirlukast in the United States, and what is its significance in drug history?: The brand name for Zafirlukast in the United States is Accolate. It holds historical significance as it was the first cysteinyl leukotriene receptor antagonist approved by the FDA.

Zafirlukast is practically insoluble in water but soluble in organic solvents like methanol.

Answer: False

Zafirlukast is practically insoluble in water but shows slight solubility in methanol; it is freely soluble in other organic solvents like tetrahydrofuran and acetone.

Related Concepts:

  • What are the key physiochemical properties of Zafirlukast?: Pure Zafirlukast is described as a fine, white to pale yellow, amorphous powder. It is practically insoluble in water but shows slight solubility in methanol and is freely soluble in solvents like tetrahydrofuran, dimethyl sulfoxide, and acetone.

Zafirlukast was approved in the US in 1996, marking it as the first leukotriene receptor antagonist.

Answer: True

Zafirlukast received US FDA approval in 1996, signifying its status as the first approved cysteinyl leukotriene receptor antagonist.

Related Concepts:

  • When was Zafirlukast approved in the United States, and what was its significance?: Zafirlukast was approved in the United States in 1996. Its approval marked a significant milestone as it was the first cysteinyl leukotriene receptor antagonist available for medical use.

Zafirlukast is exclusively used in human medicine and has no applications in veterinary care.

Answer: False

Zafirlukast does have applications in veterinary medicine, notably for the treatment of bronchial asthma in cats.

Related Concepts:

  • Is Zafirlukast used in veterinary medicine?: Yes, Zafirlukast is sometimes used in veterinary medicine for the treatment of bronchial asthma in cats.

The IUPAC name for Zafirlukast includes the term 'carbamate'.

Answer: True

The IUPAC name for Zafirlukast, Cyclopentyl 3-{2-methoxy-4-[(o-tolylsulfonyl)carbamoyl]benzyl}-1-methyl-1H-indol-5-ylcarbamate, indeed includes the term 'carbamate'.

Related Concepts:

  • What is the IUPAC name for Zafirlukast?: The IUPAC name for Zafirlukast is Cyclopentyl 3-{2-methoxy-4-[(o-tolylsulfonyl)carbamoyl]benzyl}-1-methyl-1H-indol-5-ylcarbamate.

Zafirlukast's chemical formula is C31H33N3O6S, corresponding to a molar mass of approximately 575.68 g/mol.

Answer: True

The chemical formula for Zafirlukast is C31H33N3O6S, and its molar mass is approximately 575.68 g/mol.

Related Concepts:

  • What is the chemical formula and molar mass of Zafirlukast?: The chemical formula for Zafirlukast is C31H33N3O6S, and its molar mass is 575.68 g/mol.

Zafirlukast has a melting point significantly above 200 degrees Celsius.

Answer: False

Zafirlukast has a melting point range of 138 to 140 degrees Celsius, which is not significantly above 200 degrees Celsius.

Related Concepts:

  • What is the melting point range for Zafirlukast?: Zafirlukast has a melting point range of 138 to 140 degrees Celsius (280 to 284 degrees Fahrenheit).

Zafirlukast is classified under the ATC code R03DC01.

Answer: True

Zafirlukast is classified under the Anatomical Therapeutic Chemical (ATC) Classification System with the code R03DC01.

Related Concepts:

  • What is the classification of Zafirlukast according to the Anatomical Therapeutic Chemical (ATC) Classification System?: Zafirlukast is classified under the ATC code R03DC01.

In the United Kingdom, Zafirlukast is available over-the-counter without a prescription.

Answer: False

In the United Kingdom, Zafirlukast is classified as POM (Prescription Only), meaning it is not available over-the-counter.

Related Concepts:

  • What is the legal status of Zafirlukast in the United Kingdom?: In the United Kingdom, Zafirlukast is classified as POM (Prescription only).

What is the significance of Zafirlukast (Accolate) in the history of asthma medication?

Answer: It was the first cysteinyl leukotriene receptor antagonist approved by the FDA.

Zafirlukast, marketed as Accolate, holds historical significance as it was the first cysteinyl leukotriene receptor antagonist approved by the U.S. Food and Drug Administration (FDA).

Related Concepts:

  • What is the brand name for Zafirlukast in the United States, and what is its significance in drug history?: The brand name for Zafirlukast in the United States is Accolate. It holds historical significance as it was the first cysteinyl leukotriene receptor antagonist approved by the FDA.

The source lists Zafirlukast's chemical formula as:

Answer: C31H33N3O6S

The chemical formula for Zafirlukast is C31H33N3O6S.

Related Concepts:

  • What is the chemical formula and molar mass of Zafirlukast?: The chemical formula for Zafirlukast is C31H33N3O6S, and its molar mass is 575.68 g/mol.

What is the approximate molar mass of Zafirlukast?

Answer: 575.68 g/mol

The molar mass of Zafirlukast is approximately 575.68 g/mol.

Related Concepts:

  • What is the chemical formula and molar mass of Zafirlukast?: The chemical formula for Zafirlukast is C31H33N3O6S, and its molar mass is 575.68 g/mol.

What is the ATC code for Zafirlukast?

Answer: R03DC01

The ATC code assigned to Zafirlukast is R03DC01.

Related Concepts:

  • What is the classification of Zafirlukast according to the Anatomical Therapeutic Chemical (ATC) Classification System?: Zafirlukast is classified under the ATC code R03DC01.

The source indicates that Zafirlukast is used in veterinary medicine for which condition?

Answer: Bronchial asthma in cats

Zafirlukast finds application in veterinary medicine, specifically for the treatment of bronchial asthma in feline patients.

Related Concepts:

  • Is Zafirlukast used in veterinary medicine?: Yes, Zafirlukast is sometimes used in veterinary medicine for the treatment of bronchial asthma in cats.

What is the approximate melting point range for Zafirlukast?

Answer: 138-140 °C

Zafirlukast exhibits a melting point range of approximately 138 to 140 degrees Celsius.

Related Concepts:

  • What is the melting point range for Zafirlukast?: Zafirlukast has a melting point range of 138 to 140 degrees Celsius (280 to 284 degrees Fahrenheit).

According to the source, Zafirlukast is practically insoluble in:

Answer: Water

Zafirlukast is characterized as being practically insoluble in water.

Related Concepts:

  • What are the key physiochemical properties of Zafirlukast?: Pure Zafirlukast is described as a fine, white to pale yellow, amorphous powder. It is practically insoluble in water but shows slight solubility in methanol and is freely soluble in solvents like tetrahydrofuran, dimethyl sulfoxide, and acetone.

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